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Targeting telomerase was effective at killing NRAS-mutant melanoma cells, and the impact was further enhanced when the strategy was paired with an inhibitor of mitochondrial function, according to a new study. Many melanoma patients do not benefit from new treatment options or experience disease progression because of resistance. This research advances the search for novel therapeutic strategies to treat NRAS-mutant melanoma, which is highly resistant to most therapies and associated with poor prognosis. [Source: The Wistar Institute]
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